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ferdgoodfellow -> RE: ID is not science (8/7/2008 8:47:02 AM)
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ORIGINAL: Method quote:
ORIGINAL: ferdgoodfellow But I am a loss to suggest tell-tale signs of ID other than what Dembski has already proposed: specified complexity. I don't see how that helps since there is nothing specified in biology. For example, there are (as of right now) 6 billion ways to put together a human at the genetic level. So much for specification. Also, there is no observed goal in nature so there is not a specific target that evolution is aiming for. Specified complexity fails in describing biology. Behe claims that Darwinian processes fail to completely explain biology. quote:
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Behe is a good example. He is looking at experiments already conducted by nature to see how well EB has performed. Malaria is his primary example and he claims it is the "Michelson-Morley" experiment that demonstrates the limits of the Darwinian mechanism. How did this demonstrate the limits of Darwinian mechanisms? From recollection, the malaria were under a single selective pressure away from their current state and they evolved a single solution. How does this demonstrate limits when the overwhelming selective pressure is stasis. Also, how did Behe determine that there were no other possible solutions? How did Behe determine that given different selective pressures that they could not evolve a new morphology or lifestyle? Apparently malaria is good at adapting to the different drugs science has thrown at them in past decades. There are brazillions of the little critters, their lifespan is short and they reproduce very fast. So the chances of generating beneficial mutations which will confer anbiotic resistance to the organism are great. This is a very fine demonstration of the evolutionary process, and we see it play out over the course of mere years. But there is one obstacle that malaria hasn't been able to mutate around in over 10,000 years since it appeared in humans: sickle hemoglobin. Brazillions upon brazillions of generations of malaria failed to mutate around it. This strongly suggests malaria has run into an evolutionary brick wall. Some mutations are easier dan udders, and some mutations are impossible for a given organism. And if, in this instance, EB is incapable, how then do we get off claiming that it is capable in overcoming even greater challenges. That's the gist of his reasoning. Apparently, when we get down at the molecular level and get a handle on the actual changes involved in order to achieve a beneficial mutation, we can, as in Powerball, calculate the odds. Behe borrows terminology from Jerry Coyne and Allen Orr, namely "biologically reasonable." Is it biologically reasonable to expect X to occur by reason of random mutation and natural selection at the molecular level? He offer two criteria to make this assessment: 1) Steps. The more intermediate evolutionary steps that must be climbed to achieve some bio goal without reaping a net benefit, the more unlikely a Darwinian explanation. 2) Coherence. A telltale signature of planning is the coherent ordering of steps toward a goal. Random mutation, however, is incoherent; that is, any given evolutionary step taken by a population of organism is unlikely to be connected to its predecessor.
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